B-complex vitamins are a class of water soluble vitamins which are responsible for the proper functioning of our body systems.
DATS (DIGESTION, ABSORPTION,
TRANSPORTATION AND STORAGE) OF VITAMIN-B12 Cobalamin
Vitamin B12 are also known as Cobalamin. It is a common
generic term for a group of compounds called coronoids because of their corrin
nucleus. The general compounds of cobalamin are Hydroxy cobalamin, Cyano
cobalamin, Aqua cobalamin, Nitro cobalamin, 5 deoxyadenosylcobalamin, and
methyl cobalamin.
Sources:
The only dietary source of cobalamin are the animal sources
which derived it from other microorganisms. The naturally found B12 are only
produced by microorganisms like bacteria. Plants, fungi and animals cannot
produced it by its own. If the plant contain trace amount of this vitamin than
it is probably due to any kind of contamination. Contaminated hands taking food
into mouth can also contain Vitamin B12.
The best sources of cobalamin are
sources |
Amount of Vitamin C in 100 grams |
meat |
0.6 micro gram |
egg |
1.1 micro gram |
chicken |
0.3 micro gram |
milk |
0.5 micro gram |
Poultry products, cottage cheese, yogurt and in sea sources
fishes, shell fish especially calms and oyster are the best sources of Vitamin
B12.
RDA (recommended dietary
allowances):
The recommended dietary allowances are different for the
individuals of different age groups. RDA for males, females and children’s are:
Gender |
RDA |
Men |
2.4 micro gram |
Women |
2.4 micro gram |
Children |
0.9-1.2 micro gram |
Functions of vitamin B12 in Human
body:
Vitamin B12 performs many important roles in human body
these are:
- It helps in the proper functioning of the central nervous system.
- It works with folic acid for the formation of new red blood cells.
- It acts as a co enzyme and help in body’s chemical reactions.
- Vitamin B12 also helps in the synthesis of DNA and RNA.
DATS (digestion, absorption,
transportation and storage) of vitamin C:
Injested proteins must be
released from the protein/ polypeptides to which they are bind. Salivary glands
in mouth releases hepatocorrin( R protein). This R protein binds cobalamin
releases from proteins. The R-Vitamin B12 complex are formed. This R-Vitamin
B12 complex than travels towards stomach where parietal cell releases HCl and
intrinsic factors however chief cell releases gastrin and pepsinogen. These gastric
juices are mainly responsible for the digestion of food. Thrpugh the action of
pancreatic proteases the vitamin B12 detaches from the intrinsic factor. These hepatocorrins
and If are attaches to the Vitamin B12 to protect it from bacterial use.
The absorption occurs through
out the small intestine. Most of the absorption of vitamin B12 occurs at the
terminal portion of ileum. However, the absorption occurs at the overall
section of ileum. 1-3% of absorption also occurs through passive diffusion.
After absorption cubulin receptors are present which bind the Vitamin B12
receptors and the intrinsic factor is released. Through hepatic circulation
this vitamin travels towards liver.
These cubulin recepors are of
three types: TC1, TC2 and TC3 receptors. TC1 receptor are the circulating
receptor and storage receptor which transport vitamin B12 to different body
tissues while TC2 receptor are the tissue receptor which bind vitamin B12 for
tissue usage. The function of TC3 receptor are still unknown. 80% of the
vitamin B12 transportation occurs through TC1 receptor and 20% occurs through
TC2 receptor. On the surface of ileum these receptors are present.
Hepatic portal circulation is
important for vitamin nuriture. Unlike other water soluble vitamins vitamin B12
is stored and retained for a long period of time. About 2-4 mg of the vitamin
is stored in body mainly 50% in liver. Small amount are also present in the
muscle, bones, kidney, heart, brain and spleen.
Vitamin B12 transported form
in blood is transcobalamin.
A very little urinary
excretion of vitamin B12 occurs. The protein called megalin produced in renal
tubules which cauases the reabsorption of vitamin due to which the losses are
very low. About 1.4 micro gram of cobalamin are excreted out of the body
through urine eah day.
Toxicity:
Although no clear toxicity has been reported yet. No tolerable
upper limit for Vitamin B12 intake are still established.
Megaloblastic anemia (disease caused
by Vitamin B12 deficiency):
The deficiency of Vitamin B12 leads to megaloblastic anemia.
The earlier sign and symptoms of vitamin B12 deficiency are numbness and
tingling, fatigue, muscle weakening, shortness of breath, unsteady movements,
slow healing and anemia.
Megaloblastic anemia and pernicious anemia is an autoimmune
disorder in which body produces anti bodies that attack gastric parietal cells
and there by diminished the production of IF (intrinsic factor). As due to diminished
IF production Vitamin B12 does not absorb properly. Deficiency of Vitamin B12
leads to low production of red blood cells which ultimately leads to anemia. Megaloblastic
anemia or macrocytic anemia is a form of anemia characterized by very large red
blood cells and a decrease in a number of red blood cells.
The deficiency of vitamin B12 may occur due to the following
reasons:
Dietary intake: a vegetarian person not taking fortified food
are most likely to develop Vitamin B12 deficiency.
Chronic pancreatis in which pancrease is unable to secrete
its juices due to which like other nutrients Vitamin B12 also does not
absorbed.
Bacterial over growth in peyer’s patches of ileum causes the
mal absorption.
Diet include 1 chicken breast for infants, 1 cup of plan low
fat yogurt, 1 cup of milk for men and women and 1 hard boil egg for children
may fulfill the requirements of vitamin B12.
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